Additional file 33.

Profiling the normalized (by total uniquely mapped reads) read coverage of the introns that were over-represented in the sad1 mutant relative to the wild type and SAD1-OE under the control conditions. The profiles indicate that the intron retention events in sad1 were completely or largely restored by overexpressing SAD1.

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Cui et al. Genome Biology 2014 15:R1   doi:10.1186/gb-2014-15-1-r1